LOS ANGELES TIMES - “Viruses mutate easily, so they are usually very quick to escape any therapeutic intervention,” said Juliet Morrison, a microbiologist at UC Riverside. “Any antiviral should target multiple aspects of the viral life cycle so you don’t select for resistance.”
That means finding a single pill that can disrupt not just how the coronavirus gets into cells but also how it copies itself. It should also be able to deal with the shell the virus makes to protect its precious genetic code.
“There are like 30 different proteins that SARS-CoV-2 encodes for, and all of those are potential targets,” Morrison said.